We gebruiken cookies om ervoor te zorgen dat onze site zo soepel mogelijk draait. Als je doorgaat met het gebruiken van deze site, gaan we er vanuit dat je ermee instemt.
Cardiac implantable electronic device–induced tricuspid regurgitation (CIED-induced TR) involves primary (lead-induced) and functional etiologies. The impact of left bundle branch area pacing (LBBAP) versus right ventricular pacing (RVP) on these mechanisms remains unclear.
Primary graft dysfunction (PGD) following orthotopic heart transplant (OHT) remains a common problem. Current consensus defines that PGD is diagnosed within 24 hours of surgery and subcategorized into PGD-left ventricle and PGD-right ventricle (RV). Secondary graft dysfunction suggests an etiology such as pulmonary hypertension (PH). The primary treatment for graft dysfunction is inotropic medication with or without mechanical support. The dose and duration of administration of inotropic support play roles in identifying PGD.
Primary graft dysfunction (PGD) following orthotopic heart transplant (OHT) remains a common problem. Current consensus defines that PGD is diagnosed within 24 hours of surgery and subcategorized into PGD-left ventricle and PGD-right ventricle (RV). Secondary graft dysfunction suggests an etiology such as pulmonary hypertension (PH). The primary treatment for graft dysfunction is inotropic medication with or without mechanical support. The dose and duration of administration of inotropic support play roles in identifying PGD.
Primary graft dysfunction (PGD) following orthotopic heart transplant (OHT) remains a common problem. Current consensus defines that PGD is diagnosed within 24 hours of surgery and subcategorized into PGD-left ventricle and PGD-right ventricle (RV). Secondary graft dysfunction suggests an etiology such as pulmonary hypertension (PH). The primary treatment for graft dysfunction is inotropic medication with or without mechanical support. The dose and duration of administration of inotropic support play roles in identifying PGD.